P-6

A three-member genes family is responsible for the synthesis of short chain fatty acid esters during fermentation.
JEAN-PIERRE DUFOUR (1), B. Llorente (2), B. Dujon (2), S. Kumara (1), K. Verstrepen (3) and G. Derdelinckx (3). (1) Dept Food Sci, Univ Otago, POB 56, Dunedin New Zealand; (2) Unité Génét Moléc Levures, Dept Biotech, 25 rue du Dr Roux, 75724, Paris France; (3) Centre Malt & Brew Sci, Dept Food & Microbial, KU Leuven, Kardinaal Mercierlaan 92, 3001 Heverlee, Belgium.

Ester synthesis during fermentation mainly depends on yeast ester synthesis potential, i.e. the amount of available acetyl-CoA/acyl-CoA (an essential building block for yeast cellular components) and the level of ester synthase activities, the enzymes being synthesised during the growth phase. Biochemical evidence suggests that at least five enzymes are involved in the synthesis of esters within yeast. Ester hydrolyzing activities may play a determinant role on the final beer ester levels of products such as membrane filtered beer and bottle re-fermented beer. Recently, scientists have taken advantage of the completed S. cerevisiae genome sequence database and the powerful tools of molecular biology to identify the corresponding genes and investigate the physiological role of ester synthesis. Recent rapid progress has provided insights not only into the regulation of cellular ester synthesis, but also into some general mechanisms of gene regulation. Three distinct AATase genes (ATF1, LgATF1 and ATF2), responsible for the production of acetate esters, have been cloned from different yeast backgrounds. A fourth gene, EHT1, has been described as an ethanol hexanoyl-CoA transferase. In this work, we report the effect of EHT1 disruption on the synthesis of esters. Sequence comparisons revealed that EHT1 belongs to a three-member gene family. Combination of simple, double and triple deletions did not affect growth. Disruption of one of the genes resulted in decreased levels of short chain acid esters, the decrease being accentuated with the number of disrupted genes. The results suggested an internal functional redundancy as the phenotype was increased with the number of disrupted genes. Analysis of the relationship between levels of short chain fatty acids and the corresponding ethyl esters strongly suggests that these enzymes may be involved in the removal of the toxic short chain fatty acids. Interestingly, disruption of the three-member genes family also stimulated the synthesis of the acetate esters.

Jean-Pierre Dufour. Studies: MSc., 1975-PhD., 1979 (Louvain). Research fellow (Johns Hopkins University, School of Medicine, Baltimore, MD) 1979-1981. Appointments: Catholic University of Louvain, Professor 1981-1993 (Head of the Department of Brewery and Food Industries, 1987-1993); Universidade Catolica Portuguesa, Escola superior de Biotechnologia (Porto, Portugal), visiting Professor 1989-1994; University Senghor (Alexandria, Egypt), associated Professor 1992-1995; Expert for EEC and UNIDO (Implementation of sorghum malt for the production of lager beer in Africa) (1994-1996); University of Otago, Dunedin, New Zealand, Professor (1995-present), Chairperson and Head of Department of Food Science. Expertise: Flavour science, fermentation science and technology, malting and brewing sciences, yeast biochemistry/enzymology. Active member of EBC Brewing Science Group, ASBC, IOB, Institute of Food Technology, American Chemical Society, New Zealand Institute of Food Science and Technology. Vice-president and Fellow of the New Zealand Institute of Food Science and Technology, New Zealand delegate to IUFoST.

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